A team at Nagoya University Graduate School of Medicine has found that the enzyme protein kinase N (PKN) plays a role in cardiac fibrosis linked to heart failure. In mice models, deleting PKN reduced ventricular dysfunction and tissue buildup. These findings suggest that anti-PKN therapies could potentially protect against heart failure. The research was published in Nature Communications.
A team at Nagoya University Graduate School of Medicine has found that the enzyme protein kinase N (PKN) plays a role in cardiac fibrosis linked to heart failure. In mice models, deleting PKN reduced ventricular dysfunction and tissue buildup. These findings suggest that anti-PKN therapies could potentially protect against heart failure. The research was published in Nature Communications.
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